Journal Name: International Journal of Cancer and Treatment
Article Type: Conceptual
Received date: 02 February, 2020
Accepted date: 22 July, 2020
Published date: 29 July, 2020
Citation: Absari AASA (2020) Adnan Theory for Cancer. Int J Cancer Treat Vol: 3, Issu: 2 (01-02).
Copyright: © 2020 Absari AASA . This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Abstract
For many years we have only one common concept for cancer and autoimmune diseases etiology that’s was concerned to genetic mutations all studies focused in molecular subspecies faraway from main one but actually those occurred as a continuous process for human cells metabolism in this hypothesis or concept we returned to beginning point.
Abstract
For many years we have only one common concept for cancer and autoimmune diseases etiology that’s was concerned to genetic mutations all studies focused in molecular subspecies faraway from main one but actually those occurred as a continuous process for human cells metabolism in this hypothesis or concept we returned to beginning point.
Introduction
In this concept we must be reminding chemical oxidation reaction and reduction reaction .since oxidation reaction: mean gain electrons reductive reaction: mean loss electrons we starting from this point. we must be reminding molecular biology for tumor cells and the metastasing of tumor cells to more preferring organs like(brain,liver, spleen, bone,lungs) also we must be reminding biochemical process which occurred within humancells.
Concept
Immune and cancers both are metabolic disturbances diseases, every metabolic process (oxidation or reduction) essential for all human cells .we must remember that all acids considered oxidative agents & all bases reductive agents. we must remember that body organs like lung (source for oxygen) and liver(source for sugar or hydrogen)brain (source for lipids ) lymph nodes (source for salts) we must remember the PH for most neoplastic growth is alkaline & most of ulcerative neoplasm acidic. all ulcerative neoplasm due oxidation reaction disturbances & all proliferative (growth) neoplasm due to reduction reaction disturbances, metastatic of neoplasm occurred to source of opposite ions for example ulcerative neoplasm metastasing to liver to get hydrogen and obtaining the natural stateas compensatory mechanism, and this also can prescribed for growing neoplasm witch metastasing to lung in compensatory mechanism to obtaining oxygen. (not toward preferred receptors as most of hypotheses said), so all metal earth solutions can considered bases so it’s useful for treatment of ulceration like silver but ionic form of those considered as acids and cause ulceration. Note some weak bases have properties of acids and alkaline effects like Ca(OH)2 so like those can give us ulcerative or degenerative effects and sometimes give us proliferative effects or building effects.
Conclusion
I believe in that’s most of stagnated radicals (acidic & alkaline) that considered the main cause for cancer occurrence. This can proof from why heart rarely cancerated although of it it’s approximation to breast and lung? these can be displaying the continuous & fresh metabolic activities which is essential for heart cells rather than concept that related to absence DNA mutations during cell division . another evidence most of tumors of embryonic origin( remenants of embryo ) have growing behaviour rather than destructive form which reffered to it’s abundance alkaline stagnated radicals. in opposition the epithelial cells which represent gatekeepers in which most materials passing through it (skin, colon…) still more susceptible to cancerate. so growing neoplasm can be treated by oxidative agents like nascent oxygen (fresh O²) or similar oxidative agent in opposition most of ulcerative neoplasm can be treated by reductive agents like atomic hydrogen (fresh hydrogen H²). or similar reductive agent.
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